By Alan Solomon, Deborah T. Weiss (auth.), Luigi Minetti, Giuseppe D’Amico, Claudio Ponticelli (eds.)
`The booklet may be suggested to all training nephrologists.'
Nephrology, Dialysis, Transplantation, five, 1990
`This is a well-produced and edited multi-author quantity. The illustrations are sturdy and good produced, relatively a few of the electron-micrographs. The ebook should be suggested to all training nephrologists.'
Nephrology, Dialysis, Transplantation, 5/8, 1990
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`The booklet may be instructed to all practicing nephrologists. 'Nephrology, Dialysis, Transplantation, five, 1990 `This is a well-produced and edited multi-author quantity. The illustrations are sturdy and good produced, really a few of the electron-micrographs. The publication may be suggested to all training nephrologists.
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Additional info for The Kidney in Plasma Cell Dyscrasias
Biochem and Biophys Res. Comm. 1985; 129: 701-6. Shirahama T, Skinner M, Cohen AS, Gejyo F, Arakawa M, Suzuki M, Hirasawa Y. Histochemical and immunohistochemical characterization of amyloid associated with chronic hemodialysis as J32-microglobulin. Lab Invest 1985; 53: 705-9. Gorevic PD, Munoz PC, Casey TT, DiRaimondo CR, Stone WJ, Prelli FC, Rodrigues MM. Poulik MD, Frangione B. Polymerization of intact J32-mieroglobulin in tissue causes amyloidosis in patients on chronic hemodialysis. Proc Natl Acad Sci USA 1986; 83: 7908-12.
Proc Nat! Acad Sci USA 1981; 78: 7096-100. 6. Caligaris-Cappio F, Janossy G, Bergui L, Tesio L, Pizzolo G, Malavasi F, Chilosi M, Campana D, van Camp B, Gavosto F. Identification of malignant plasma cell precursors in the bone marrow of multiple myeloma. J Clin Invest 1985; 76: 1243-51. 7. Epstein 1, Barlogie B, Alexanian R. Phenotype heterogeneity in multiple myeloma with aneuploid plasma cells. Proceedings ASCO 1987; Vol. 6, Abstr 756. 8. Latreille J, Barlogie B, Johnston D, Maxwell B, Drewinko B, Alexanian R.
It is furthermore anticipated that studies of B cell growth and differentiation factors and their receptors will help 29 unravel the mechanisms of uncontrolled plasma cell growth (possibly linked to alterations of cellular oncogenes such as c-myc and bcl-l) and thus provide new targets for both cytotoxic and biologic therapy . Acknowledgement This work was supported by CA37161 and CA28771 from the National Cancer Institute, Bethesda, Md 20203. References 1. Durie BMG, Salmon SE. A clinical staging system for multiple myeloma.