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Reinforcement of the afterdischarge of the callosal potentials by cortical potentials evoked by stimulation of the n. ventralis posterior. The top three traces are control recordings of the callosal potentials. In each of the lower traces, the first response is the cortical evoked potential from stimulation of the n. ventralis posterior. Following it are the sequence of the callosal potentials. In order for the facilitation of the amplitude of the slow component of the test response to appear, both the conditioning and the test stimuli must be of sufficient intensity.

Ventralis posterior, and the test stimuli being the same as the above; an increase in the slow component of the callosal potential could be seen (Fig. 5b, trace 3). Next, three stimuli were used, the first evoking the callosal potential, the second and third being the same as for the above-mentioned conditioning and test responses, respectively; it was apparent that following the increase in amplitude of the potential evoked by stimulation of the n. ventralis posterior, the slow component of the test response clearly increased (Fig.

0 sec Fig. 1. Time factor of the excitability change following stimulation of the homotopic point on the contralateral cerebral cortex. A. Recordings of potentials, t indicates the control recordings of evoked potentials from stimulation of the superficial radial nerve (test response). In the recordings in Groups 1, 2,3, and 4, the first response is the callosal potential resulting from stimulation of the homotopic point of the contralateral cortex (conditioning response), and the following one is the sequence of the test response, led from an electrode in the middle of the region yielding the test response.

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